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📝 “What are the Different Types of Facial Paralysis?”
Published 1Â year, 1Â month ago
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Quick Review #265 - #pathology #oralpathology #doctorgallagher #oralsurgery #oralsurgeon #dentist #dentistry #dental #facialparalysis #bellspalsy
- 2.6.25
Facial paralysis is classified based on location, onset, progression, and cause.
- ​ Central vs. Peripheral Paralysis
- ​ Central (UMN - brain lesion): Forehead is spared, contralateral weakness; seen in stroke, multiple sclerosis (MS), brain tumors.
- ​ Peripheral (LMN - facial nerve lesion): Entire face affected, ipsilateral weakness; seen in Bell’s palsy, Ramsay Hunt syndrome, Lyme disease, trauma, parotid tumors (De Diego-Sastre et al., 2016).
- ​ Acute vs. Chronic Paralysis
- ​ Acute (<72 hrs onset): Bell’s palsy, stroke, Ramsay Hunt, trauma; sudden weakness, pain, hyperacusis, taste loss.
- ​ Chronic (>3 months): Parotid tumors, neurosarcoidosis, Melkersson-Rosenthal syndrome; gradual worsening, mass effect (Fattah et al., 2014).
- ​ Congenital vs. Acquired Paralysis
- ​ Congenital: Moebius syndrome, congenital facial nerve palsy, hemifacial microsomia; present at birth, craniofacial defects possible.
- ​ Acquired: Bell’s palsy, stroke, tumors, infections; develops later due to underlying pathology (Eviston et al., 2015).
- ​ Complete vs. Incomplete Paralysis
- ​ Complete: Total loss of voluntary movement, inability to close the eye, severe Bell’s palsy, trauma, full nerve damage.
- ​ Incomplete (paresis): Partial movement remains, seen in mild Bell’s palsy, partial nerve compression.
- ​ Unilateral vs. Bilateral Paralysis
- ​ Unilateral: Bell’s palsy, stroke, Ramsay Hunt, trauma, parotid tumors; one-sided weakness.
- ​ Bilateral: Guillain-Barré syndrome (GBS), Lyme disease, neurosarcoidosis, Moebius syndrome, Melkersson-Rosenthal syndrome; symmetrical facial weakness, systemic symptoms.
- ​ Recurrent vs. Progressive Paralysis
- ​ Recurrent: Melkersson-Rosenthal syndrome, MS, idiopathic facial palsy; episodes of weakness, recovery in between.
- ​ Progressive: Tumors, chronic infections, neurodegenerative diseases; gradual worsening, systemic involvement.
References:
- ​ Shaina. (2024, July 9). Bell’s palsy: What is it and how to treat it? Gulf Physio.
- ​ De Diego-Sastre, J. I., Prim-Espada, M. P., & Fernández-GarcĂa, F. (2016). The epidemiology of Bell’s palsy. Revue de Laryngologie - Otologie - Rhinologie, 137(4-5), 173-176.
- ​ Eviston, T. J., Croxson, G. R., Kennedy, P. G. E., Hadlock, T., & Krishnan, A. V. (2015). Bell’s palsy: Aetiology, clinical features, and multidisciplinary care. Journal of Neurology, Neurosurgery & Psychiatry, 86(12), 1356-1361
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